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The Journal of Urology
Volume 181, Issue 6
, Pages 2566-2572
, June 2009
Renal Phosphate Control as a Reliable Predictive Factor of Stone Recurrence
References
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- Inappropriate phosphate excretion in idiopathic hypercalciuria: the key to a common cause and future treatment?. J Clin Pathol. 1996;49:881
- “Idiopathic” hypercalciuria and hereditary hypophosphatemic rickets (Two phenotypical expressions of a common genetic defect). N Engl J Med. 1987;316:125
- . Frequency of renal phosphate leak among patients with calcium nephrolithiasis. Kidney Int. 2001;60:272
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- Sodium-hydrogen exchanger regulatory factor-1 interacts with mouse urate transporter 1 to regulate renal proximal tubule uric acid transport. J Am Soc Nephrol. 2007;18:1419
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- . Metabolic evaluation of stone disease patients: a practical approach. J Endourol. 1999;13:669
- . Thiazides reduce brushite, but not calcium oxalate, supersaturation, and stone formation in genetic hypercalciuric stone-forming rats. J Am Soc Nephrol. 2005;16:417
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- . Targeted inactivation of Npt2 in mice leads to severe renal phosphate wasting, hypercalciuria, and skeletal abnormalities. Proc Natl Acad Sci U S A. 1998;95:5372
- . Regulation of Na/Pi transporter in the proximal tubule. Annu Rev Physiol. 2003;65:531
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Supported by the Korea Science and Engineering Foundation Grant funded by the Korean government (MEST) (PT-ERC).
For another article on a related topic see page 2767.
PII: S0022-5347(09)00271-7
doi: 10.1016/j.juro.2009.01.101
© 2009 American Urological Association. Published by Elsevier Inc. All rights reserved.
« Previous
Next »
The Journal of Urology
Volume 181, Issue 6
, Pages 2566-2572
, June 2009

